Let’s review some recent history and background on the present pandemic situation.
(This is not an opinion for or against any political decision – it is simply information from academic/medical journals and government data!)

Scientists around the world have been searching for viruses that might cause pandemics. Why? – Because some of the past virus outbreaks were close to being called a pandemic. They wanted to know what the potential might be for a more rampant virus.

WHAT was known following the 2003 SARS outbreak:
Diverse SARS-like coronaviruses (SL-CoVs) have now been reported from bats in China, Europe and Africa but none is considered a direct progenitor of SARS-CoV because of their phylogenetic disparity from this virus and the inability of their spike proteins to use the SARS-CoV cellular receptor molecule, the human angiotensin converting enzyme II (ACE2)
https://www.nature.com/articles/nature12711#ref-CR5

UNTIL further research was conducted – specifically in China:
Peter Daszak and colleagues identify two novel coronaviruses from Chinese horseshoe bats that are closely related to severe acute respiratory syndrome coronavirus (SARS-CoV), the cause of a pandemic during 2002 and 2003.

They also isolate a live virus from these bats that has high sequence identity to SARS-CoV and that can infect human cells using ACE2, the same receptor that is used by SARS-CoV. The results provide the strongest evidence to date that horseshoe bats are natural reservoirs of SARS-CoV.

Here we report whole-genome sequences of two novel bat coronaviruses from Chinese horseshoe bats (family: Rhinolophidae) in Yunnan, China: RsSHC014 and Rs3367. These viruses are far more closely related to SARS-CoV than any previously identified bat coronaviruses, particularly in the receptor binding domain of the spike protein. Most importantly, we report the first recorded isolation of a live SL-CoV (bat SL-CoV-WIV1) from bat faecal samples in Vero E6 cells, which has typical coronavirus morphology, 99.9% sequence identity to Rs3367 and uses ACE2 from humans, civets and Chinese horseshoe bats for cell entry.

Our results provide the strongest evidence to date that Chinese horseshoe bats are natural reservoirs of SARS-CoV, and that intermediate hosts may not be necessary for direct human infection by some bat SL-CoVs. They also highlight the importance of pathogen-discovery programs targeting high-risk wildlife groups in emerging disease hotspots as a strategy for pandemic preparedness.

Thus, the Horseshoe Bat in China – does contain a SARS virus that has the ability to attack humans – but how do they do this? – By attaching to ACE2 cells in the human body.

WHAT is ACE?:

Angiotensin-converting enzyme (ACE) inhibitors help relax your veins and arteries to lower your blood pressure. ACE inhibitors prevent an enzyme in your body from producing angiotensin II, a substance that narrows your blood vessels. This narrowing can cause high blood pressure and force your heart to work harder. Angiotensin II also releases hormones that raise your blood pressure. In addition to high blood pressure, ACE inhibitors prevent, treat or improve symptoms in conditions such as: Coronary artery disease, Heart failure, Diabetes, Certain chronic kidney diseases, Heart attacks, Scleroderma — a disease that involves hardening of the skin and connective tissues, Migraines
In rare cases, particularly for black people, women and smokers, ACE inhibitors can cause some areas of the tissues to swell (angioedema). If swelling occurs in the throat, it can be life-threatening.

MORE on ACE2:

Angiotensin-converting enzyme 2; Carboxypeptidase which converts angiotensin I to angiotensin 1-9, a peptide of unknown function, and angiotensin II to angiotensin 1-7, a vasodilator. Also able to hydrolyze apelin- 13 and dynorphin-13 with high efficiency.

https://string-db.org/network/9606.ENSP00000389326

SO – if people taking ACE2 inhibitors are disrupting the natural body conversion of ACE2 – there is no telling what has been happening to the body when the SARS-2 Covid-19 attacks the ACE2 cells that are suppressed by medication. (Note the above statement about “peptide of unknown function”)

AND – there were studies completed just prior to the official pandemic in the US –
“Since patients treated with ACEIs and ARBS will have increased numbers of ACE2 receptors in their lungs for coronavirus S proteins to bind to, they may be at increased risk of severe disease outcomes due to SARS-CoV-2infections,” explains Diaz.
Diaz writes, this hypothesis is supported by a recent descriptive analysis of 1,099 patients with laboratory-confirmed COVID-19 infections treated in China during the reporting period, December 11, 2019, to January 29, 2020. This study reported more severe disease outcomes in patients with hypertension, coronary artery disease, diabetes and chronic renal disease. All patients with the diagnoses noted met the recommended indications for treatment with ACEIs or ARBs. Diaz says that two mechanisms may protect children from COVID-19 infections — cross-protective antibodies from multiple upper respiratory tract infections caused by the common cold-causing alpha coronaviruses, and fewer ACE2 receptors in their lower respiratory tracts to attract the binding S proteins of the beta coronaviruses.
https://www.sciencedaily.com/releas…/2020/…/200323101354.htm

And, further study that showed how children have higher levels of ACE than elderly – https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6192041/

While many MDs and scientists have varying opinions about what is causing the COVID-19 to be such a terrible virus – one thing is true:

UPDATE from CDC – Sept 3, 2020 – https://covid.cdc.gov/covid-data-tracker/#demographics

Cases of Covid-19:
0-50 age: 2,794,132
Deaths: 7045
.0025%

NOTE: (Is this why schools are canceled??)
Under age 30: 1,349,878
Deaths: 776
.00574%

Over age 50: 1,623,480
Deaths: 130,083
8.01%

FYI – in 2017 (ages 10-24) Death Rates due to Suicide and Homicide (both preventable by society)
Suicide: 6,679
Homicide: 5083
https://www.cdc.gov/nchs/products/databriefs/db352.htm#ref5